Why does Stomach growl when hungry?
Although a rumbling stomach is usually heard and is associated with hunger and the absence of food in the stomach, it can occur at any time, on an empty or full stomach. In addition, growls not only come from the stomach, but are also often heard from the small intestine. Growling is more often associated with hunger because it is usually louder when the stomach and intestines are empty so the organ contents don't muffle the sound.
This growl has been such a hobby for so many years that the ancient Greeks came here with an interesting name instead: borborygmi (plural of borborygmus). The etymology of the time period depends on the onomatopoeia; it is trying to put the roar into words. Borborygmi without hesitation interprets it as "rumbling."
The physiological foundation of this rumble requires training the muscles in the stomach and small intestine. In general, the gastrointestinal tract is a tube opening that extends from the mouth to the anus and the septa are principally composed of layers of striated muscle. When the partition is activated and compresses the contents of the duct to combine and propel food, fuel and fluids through the stomach and small intestine, it produces a rumbling sound. This stretching of the muscle partitions is called peristalsis and requires the contraction rings to slide aborally (away from the oral cavity) toward the anus several inches at a time.
This peristaltic wave technology results from sensible electrical rhythmic fluctuations in net muscle cells, which, all corresponding different prerequisites, will cause the muscles to contract. These fluctuations are referred to as basic electrical rhythms (BER) and are the end result of inherent recreation of the enteric fear system, which is determined in the intestinal partitions. BER moves the muscle cells of the stomach and small intestine to propel at a normal rhythm (three and 12 times per minute, respectively), in a manner that is comparable to, but slower than, the rhythm of the heart muscle in the heart. Autonomic anxiety devices and hormonal elements can modulate this BER.
Although peristaltic pressure and pressure will usually increase in the presence of food, activity will also increase after the stomach and small intestine have been empty for about two hours. In the latter case, receptors in the stomach partition undergo the absence of food, eliciting electric wave reflex technology (migrating myoelectric complexes, or MMCs) in the enteric fear system. These MMCs travel along the stomach and small intestine and cause starvation contractions. Such starvation contractions begin in the antrum, or area of descent, of the stomach and spread along the size of the complete intestine, sweeping into the terminal ileum. They clean any and all stomach contents including mucus, last food ingredients and bacteria and keep them from collecting in one place.
Contractions also produce vibrations and rumbling sounds associated with hunger. Hunger contractions may also last for 10 to 20 minutes as soon as they begin, and then repeat every one to two hours until the next meal is swallowed. This is no longer synonymous with hunger pains, which begin 12 to 24 hours after the rest of a meal and can also continue for several days before subsides. (Perhaps such pain is important in the sensation of hunger that induces the animal to eat.) Low blood sugar increases this activity, which can also be caused by intravenous infusion of the hormone motilin. After eating, MMC subsided.
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